Of the several varieties of hyperthyroidism, one of the most common is Graves disease. Graves disease is an autoimmune disease in which thyroid-stimulating hormone (TSH) receptor antibodies bind to and stimulate the thyroid gland, causing the excessive secretion of thyroxine (T₄) or triiodothyronine (T₃) or both, resulting in the clinical manifestations of thyrotoxicosis.1,2,3,4,5 and 6 Graves disease has fascinated clinicians because of its possible relationship to stress, its unusual and varied manifestations (e.g., eye involvement, pretibial myxedema), and its unpredictable course, characterized by relapses and exacerbations.